Yes- living cells.
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Life After Ventilators Can Be Hell for Coronavirus Survivors
Some never fully recover, and those who do often must relearn basic skills such as walking and swallowing.
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I am slap dab in between Ga and NC… eh who knows??
Seems like Ga is double of NC… and still rising… while SC is even lower than NC…
With ATL CHAR CHAS and Myrtle Beach, one would think SC would be ate slap up…
Even MB is lower than my town…
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GA has something NC does not… “ATL”, one of the busiest airports in the world, with untold infected parties arriving on international flights for how-knows-long before things started getting shut down…
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Explain Charlotte… 
both are mega towns… and both are exactly 2 hours from me… Columbia is 3… but it is smaller than either ATL or CHAR… even here, we have international flights, Phil… do not forget, we have people coming from Germany and Sweden all the time, with BMW, Michegan and Lockheed… SC is a manufacturing state, mostly textiles and car/carp parts now days… but we were the place for cotton, tea and tobacco…
My state is surrounded by infected…
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So defensive.
Hartsfield–Jackson Atlanta International Airport has been the world’s busiest airport every year since 2000
But CLT is 34th so don’t feel too bad. And if it helps, I’ve been there plenty. I usually stop at the Five Guys by the gate I typically get. It’s a nice airport.
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I prefer to fly out of GSP as it is a lot smaller… and because it’s cheeaper to connect to larger flights later. 
Yeah I know… I just hope there is no big flair ups… by June 1… everything should be reopening… then we need to keep an eye out in the fall and spring… it’s not over by a long shot.
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(The Guardian, April 24, 2020):
Coronavirus has been detected on particles of air pollution by scientists investigating whether this could enable it to be carried over longer distances and increase the number of people infected. …
… Two other research groups have suggested air pollution particles could help coronavirus travel further in the air. A statistical analysis by Setti’s team suggests higher levels of particle pollution could explain higher rates of infection in parts of northern Italy before a lockdown was imposed, an idea supported by another preliminary analysis. The region is one of the most polluted in Europe. …
… Previous studies have shown that air pollution particles do harbour microbes and that pollution is likely to have carried the viruses causing bird flu, measles and foot-and-mouth disease over considerable distances.
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What “magical shields” exist over SC ? Could it possibly be a case of limited testing conducted ? 
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Edit; DO NOT INGEST DISINFECTANTS !
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For those among His Faithful Flock too macho to ever be caught “sucking on them Gina Sticks”:
Source: https://i.pinimg.com/originals/89/f0/6f/89f06f2ab627d316f61ddf52c88d14d8.jpg
Source: https://i.insider.com/50b39ed4eab8ea4f6e00001a?width=400
Bestus Potus-Apocalyptic Living - Thanks To Our Bleeding Edge Scientific Research !!!
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LEAVE JUULIA ALONE !
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(Business Insider, April 16, 2020):
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Just when you thought that it (might, hopefully, please) be “safe to go back in the water” (someday):
Non-human primates vaccinated with modified vaccinia Ankara (MVA) virus encoding full-length SARS-COV spike glycoprotein and challenged with the SARS-CoV virus had lower viral loads but suffered from acute lung injury due to antibody enhancement.[3] A study of 29 hospitalized and subsequently recovered Coronavirus disease 2019 (COVID-19) patients after severe acute respiratory syndrome coronavirus 2 (SARS-COV-2) infection found that 100% of patients had anti-spike antibodies.[4] Anti-spike IgG correlated linearly with age and LDH (a biomarker of disease severity often elevated in cytokine release syndrome). Antibody-dependent enhancement has been observed in both severe acute respiratory syndrome (SARS) and Middle East respiratory syndrome (MERS) animal models allowing the respective viruses to enter cells expressing Fc𝛾R including myeloid lineage cells.[5]
Antibody-dependent enhancement of acute lung injury has been documented in animal models of both SARS and MERS. Rabbits intranasally infected with MERS-COV developed a pulmonary infection characterized by viremia and perivascular inflammation of the lung, and an antibody response that lacked neutralizing antibodies.[6] The rabbits developed more severe lung disease on re-exposure to MERS-COV, and developed neutralizing antibodies after reinfection.[6] In SARS, mice vaccinated with four types of vaccines against SARS-COV, as well as those infected with SARS-COV itself, developed neutralizing antibodies.[7] Mice were then challenged with live SARS-COV, upon which all developed an immunopathologic-type lung disease, although none had detectable virus two days after challenge and were protected compared to control.[7] The development of immunopathology upon exposure has been a major challenge for coronavirus vaccine development[7] and may similarly impact SARS-COV-2 vaccine research.
Source: https://en.wikipedia.org/wiki/Antibody-dependent_enhancement#In_coronavirus_infection
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Good Gawd, Just Shoot Me Now ! - (OR) - Whatever You Do, Just Don’t Send Me The Laundry Bill !
Spot, a four-legged robot, is being tested at Boston’s B&W Hospital as a way to treat COVID-19 patients
“Spot doesn’t need to wear a mask or gown.”
"Meet ‘Spot:’ The Robot That Could Help Doctors Remotely Treat COVID-19 Patients"
I would be willing to bet that the annual General Hospital Employee Picnic is a pretty weird affair ? 
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(Josh Bloom, ACSH, April 23, 2020):
“What The Apparent Failure Of Remdesivir In Its First Controlled Trial Means And Doesn’t Mean”
.
(Josh Bloom, ACSH, April 24, 2020):
“If Remdesivir Really Fails There May Be No Treatment For Coronavirus”
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(Ars Technica, April 24, 2020):
Frustrated statisticians and epidemiologists took to social media this week to call out substantial flaws in two widely publicized studies trying to estimate the true spread of COVID-19 in two California counties, Santa Clara and Los Angeles. … it’s important to note that neither of them have been published in a scientific journal, nor have they gone through standard peer-review for scientific vetting. Instead, they have been posted online in draft form … The findings seemed to support minority arguments that COVID-19 may be no worse than seasonal flu (a leading cause of death in the US) and that the restrictive mitigation efforts currently strangling the economy may be unnecessary. In fact, three researchers who co-authored the new studies have publicly made those exact arguments. …
… Because diagnostic testing has been so limited in the US and many COVID-19 cases appear to present with mild or even no symptoms, researchers expect the true number of people who have been infected to be much higher than we know based on confirmed cases. There is no debate about that. But just how much higher is the subject of considerable debate. The researchers went about their studies by recruiting small groups of residents and testing their blood for antibodies against SARS-CoV-2. Antibodies are Y-shaped proteins that the immune system makes to target specific molecular foes, such as viruses. If a person has antibodies that recognize SARS-CoV-2 or its components, that suggests the person was previously infected.
A lot of good detailed information (about research methods, limitations, criticisms) follows in the article.
(The Guardian, April 24, 2020):
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