@daath
I know I’m hoping for something effective soon. I’m also hoping that Australia’s infection rate doesn’t start to climb. I’m going to guess but I think countries will horde what proper PPE they have if it does? I think Australia’s infection rate will be both a litmus test and a boost in incentive not to help other countries. I really do hope we’ve seen the worst of it.
After finally having time to view the video, my take away from it are a few questions out of curiosity:
Did they find the proper doses when doing the study? (a previous video I showed suggested there could be to much\little zinc in a cell).
What’s the time frame the treatment takes?
Does using this ruin any other choices?
I guess it’s obvious by now it’s not incredibly effective by this study. However, if it’s the highest probability of success treating the disease, I have to go with it as to apposing it.
In case you may have missed some of my posts up-thread. Pulmonary alveoli need small amounts of Vitamin A related Carotenoids for normal functionality. An adequate Vitamin D3 intake (which is around => 2,000 IU daily for adults) has recently been proposed as something that (may, possibly) assist the immune-system in relation to SARS-CoV-2 infection resistance. I found some articles/papers (regarding Vitamins D) - but have refrained from posting them (the reason for the caution stated directly below).
In both cases it is entirely possible to ingest way too much of both (particularly Vitamins D, which are essentially “cumulative” in one’s system, due to the long ~3 week plasma half-live of the Calcidiol that the Liver makes from ingested Vitamins D). D3 is the variety that skin exposure to sunlight generate and most OTC supplements contain. Vitamin D2 does not appear to be clinically any better than Vitamin D3.
The (for me, and as reported by Sanjay Gupta MD) obvious problem of N95 type masks (especially when properly fitted) is the restricted ability to inspire new air in order to breathe - even at rest, much less when engaging in any even trivial amounts of ohysical movements and exertions. Thus, the very (cardio-pulmonary) compromised risk groups who are much more likely to suffer severe internal organ damages/failure (or die) from the COVID-19 disease resulting from infection with the SARS-CoV-2 viral microbe would be dubiously placed at notable and immediate risk for adverse or fatal cardio-vascular events. The “surgical” types accept particles/droplets up to a whopping 100 Microns size, up to 100 times larger than droplets found to carry SARS-CoV-2 microbes, and around 1,000 times the physical size of the microbes. (Only) useful to limits wearer’s “emanations” (somewhat). Neither “surgicals” nor “N95s” equal very effective protection from microbes. It’s just too complex to follow the strict “protocols”.
I just got my latest MIT Technology Review for anyone interested in a good long read. @Raven-Knightly I think you said you enjoyed reading these. There’s a lot to read in this one. I hope you get into it and start sharing because I can’t even tackle this right now. Too much laundry to do, lol. It even covers “Plandemic”.
There is growing evidence that the virus causes a far greater array of symptoms than was previously understood. And that its effects can be agonisingly prolonged: in Garner’s case for more than seven weeks. … According to the latest research, about one in 20 Covid patients experience long-term on-off symptoms. It’s unclear whether long-term means two months, or three or longer. … Many Covid patients do not develop a fever and cough. Instead they get muscle ache, a sore throat and headache. … tracked 15 different types of symptoms, together with a distinct pattern of “waxing and waning”. “I’ve studied 100 diseases. Covid is the strangest one I have seen in my medical career,” Spector says.
@Raven-Knightly,
I totally agree. The only way to move enough air past such a dense material would have to require surface area to do.something folded like an accordion (automotive air cleaner. is the only way that level of filtration can exist and still breath. Surface are is key. That’s why I say reusable respirators.They hold the key to proper filtration without the restrictions the current N95 masks do.
Thank you. Having a friend in the medical profession I found this a helpful read. I need to check in on her. My God how the world is changing before our very eyes.
That SARS-CoV-2 appears to be able (even so far in a small known number of children) to evidently asymptomatic-ally infect and induce relevant antigen-specific antibodies - then (several weeks later) trigger life-threatening conditions surrounding “immune-systems gone wild” is genuinely concerning - as it may be that adults can (also) experience similar (or more generally related) episodes after a point where they, like these cases involving children, have tested negative for antigen (using PCR testing).
Note that the “swabbing” only takes place well behind the nose and in the throat. While these mucus-membranes (along with those that line our intestinal tracts) are thought to be the most likely (given the antigens observed highest “ACE2 receptor binding affinities”) - the absence of measurable antigen in those locations does not rule out persistence of the viral antigen in numerous other bodily organs and tissues. With regards to hoped for mitigation and vaccination medications, the SARS-CoV2 antigen has been posited to be able to assume molecular conformations that “hide” the key target “spike protein” (see this previously posted info up-thread) - the same “spike protein” that diagnostic tests as well as medications will attempt to recognize and target. Numerous viruses are not eliminated, but persist in various forms for very long time periods. The success of compromised immune-systems in mounting a viable defense (either due to age, disease-states, or plethora of “biological” drugs all the rage these days, which intentionally act as immune-suppression agents) likely varies, and such things may possibly lead to ongoing post-infection complications (that will likely take many years to clinically characterize).
Despite the daily updates on number of cases, hospital admissions, and deaths around the world and the increasing number of hospital-based case series, some of the fundamental information about how severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2) spreads in the population and who is really at risk of both infection and severe consequences is still missing. …
… Of 3802 tests, 587 (15·4%) were positive for SARS-CoV-2. Prevalence of infection was less than 5% in patients younger than 18 years (23 patients were positive [4·6%] of 499 tested) but almost four times as high in people aged 40 years or older (480 [18·2%] of 2637). After adjustment for other factors, infection risk was higher among men than women (odds ratio [OR] 1·55 [95% CI 1·27–1·89]), in black people than white people (OR 4·75 [2·65–8·51]), and in people with obesity than normal-weight people (1·41 [1·04–1·91]). Infection risk was also higher in those living in more deprived or in urban versus rural locations. Surprisingly, household size did not significantly affect infection risk. Among chronic comorbidities examined, only those with chronic kidney disease had an increased risk of infection, whereas the risk in active smokers was around half that observed in never smokers. …
… this study suggests that sex differences in poor outcomes from COVID-19 are at least in part related to differential infection susceptibility. The role of ethnicity in greater susceptibility and poorer prognosis is a growing concern and deserving of further study. It seems that most comorbidities (except chronic kidney disease), although important for predicting prognosis, do not have a major part in susceptibility to infection. Regarding the results on smoking, it is likely that they could reflect consulting patterns and higher rates of non-infectious cough among smokers than non-smokers. Smoking seems important as a risk factor for poor prognosis, but studies are conflicting, and the association merits further investigation. The one major modifiable risk factor is obesity, which presents a double problem of increasing susceptibility to infection, as well as the risk of severe consequences.
The graphic charts below were previously published on May 4, 2020:
14 days later, the estimate of US deaths by August 4, 2020 has increased by 35,321 to latest (May 18, 2020) estimate of 196,642 deaths [ 121,081 - 338,950 ], increasing in total estimated number (on average) by 2,523 deaths every day. That is a +21.90% increase in estimated deaths (since May 4, 2020). In the period of March 4, 2020 - August 4, 2020, an average of 141,854 people are estimated to have become infected every day (that’s 5,911 people infected per hour, or 99 people per minute).
